Nicoletti, Natália Fontana <br/><br/>
Mechanisms involved in kinin-induced glioma cells proliferation: the role of ERK1/2 and PI3K/Akt pathways.  [electronic resource] 

 -  Journal of neuro-oncology  Nov 2014 
 - 235-44 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1573-7373 
<br/><br/><label>Standard No.: </label>10.1007/s11060-014-1549-4  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Apoptosis<br/>Blotting, Western<br/>Bradykinin--analogs & derivatives<br/>Bradykinin B1 Receptor Antagonists--pharmacology<br/>Bradykinin B2 Receptor Antagonists--pharmacology<br/>Cell Proliferation<br/>Dioxoles--pharmacology<br/>Flow Cytometry<br/>Glioma--drug therapy<br/>Humans<br/>Mitogen-Activated Protein Kinase 1--genetics<br/>Mitogen-Activated Protein Kinase 3--genetics<br/>Phosphatidylinositol 3-Kinases--genetics<br/>Phosphorylation--drug effects<br/>Proto-Oncogene Proteins c-akt--genetics<br/>RNA, Messenger--genetics<br/>Real-Time Polymerase Chain Reaction<br/>Receptor, Bradykinin B1--chemistry<br/>Receptor, Bradykinin B2--chemistry<br/>Reverse Transcriptase Polymerase Chain Reaction<br/>Sulfonamides--pharmacology<br/>Tumor Cells, Cultured