Allam, Ramanjaneyulu <br/><br/>
Mitochondrial apoptosis is dispensable for NLRP3 inflammasome activation but non-apoptotic caspase-8 is required for inflammasome priming.  [electronic resource] 

 -  EMBO reports  Sep 2014 
 - 982-90 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1469-3178 
<br/><br/><label>Standard No.: </label>10.15252/embr.201438463  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Apoptosis--genetics<br/>Autophagy--genetics<br/>Bone Marrow Cells--metabolism<br/>Carrier Proteins--biosynthesis<br/>Caspase 8--biosynthesis<br/>Cells, Cultured<br/>Peptidyl-Prolyl Isomerase F<br/>Cyclophilins--antagonists & inhibitors<br/>Humans<br/>Inflammasomes--metabolism<br/>Interleukin-1beta--biosynthesis<br/>Mitochondria--metabolism<br/>Mitophagy--genetics<br/>NLR Family, Pyrin Domain-Containing 3 Protein<br/>Toll-Like Receptors--metabolism<br/>Ubiquitin-Protein Ligases--genetics