Sun, Bei Bei <br/><br/>
Silencing of JMJD2B induces cell apoptosis via mitochondria-mediated and death receptor-mediated pathway activation in colorectal cancer.  [electronic resource] 

 -  Journal of digestive diseases  Sep 2014 
 - 491-500 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1751-2980 
<br/><br/><label>Standard No.: </label>10.1111/1751-2980.12166  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Apoptosis--genetics<br/>Caspases--metabolism<br/>Colorectal Neoplasms--genetics<br/>Cytochromes c--metabolism<br/>Down-Regulation<br/>Endoplasmic Reticulum Chaperone BiP<br/>Endoplasmic Reticulum Stress--physiology<br/>Gene Silencing<br/>Humans<br/>Jumonji Domain-Containing Histone Demethylases--genetics<br/>Mitochondria--physiology<br/>Neoplasm Proteins--metabolism<br/>Proto-Oncogene Proteins c-bcl-2--biosynthesis<br/>RNA Interference<br/>Receptors, Death Domain--physiology<br/>Tumor Cells, Cultured<br/>bcl-2 Homologous Antagonist-Killer Protein--metabolism<br/>bcl-2-Associated X Protein--metabolism