Cai, Benzhi <br/><br/>
Deletion of FoxO1 leads to shortening of QRS by increasing Na(+) channel activity through enhanced expression of both cardiac NaV1.5 and β3 subunit.  [electronic resource] 

 -  Journal of molecular and cellular cardiology  Sep 2014 
 - 297-306 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1095-8584 
<br/><br/><label>Standard No.: </label>10.1016/j.yjmcc.2014.06.006  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Action Potentials--physiology<br/>Animals<br/>Cell Nucleus--metabolism<br/>Connexin 43--genetics<br/>Electrocardiography<br/>Forkhead Box Protein O1<br/>Forkhead Transcription Factors--deficiency<br/>Gene Expression Regulation<br/>Heart Ventricles--metabolism<br/>Humans<br/>Mice<br/>Mice, Knockout<br/>Myocardial Infarction--genetics<br/>Myocytes, Cardiac--metabolism<br/>NAV1.5 Voltage-Gated Sodium Channel--genetics<br/>Patch-Clamp Techniques<br/>Primary Cell Culture<br/>Signal Transduction<br/>Voltage-Gated Sodium Channel beta-3 Subunit--genetics