Lan, Xiqian <br/><br/>
APOL1 risk variants enhance podocyte necrosis through compromising lysosomal membrane permeability.  [electronic resource] 

 -  American journal of physiology. Renal physiology  Aug 2014 
 - F326-36 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, Non-P.H.S. 
<br/><br/><label>ISSN: </label>1522-1466 
<br/><br/><label>Standard No.: </label>10.1152/ajprenal.00647.2013  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>4,4'-Diisothiocyanostilbene-2,2'-Disulfonic Acid--pharmacology<br/>Actins--metabolism<br/>Black or African American--ethnology<br/>Apolipoprotein L1<br/>Apolipoproteins--genetics<br/>Cells, Cultured<br/>Chloride Channels--antagonists & inhibitors<br/>Chloroquine--pharmacology<br/>Genetic Predisposition to Disease--ethnology<br/>Genetic Variation--genetics<br/>Glomerulosclerosis, Focal Segmental--ethnology<br/>Humans<br/>Lipoproteins, HDL--genetics<br/>Lysosomes--physiology<br/>Necrosis--physiopathology<br/>Permeability<br/>Podocytes--drug effects