TY  - GEN
AU  - Jiang,Haiying
AU  - Cheng,Xian Wu
AU  - Shi,Guo-Ping
AU  - Hu,Lina
AU  - Inoue,Aiko
AU  - Yamamura,Yumiko
AU  - Wu,Hongxian
AU  - Takeshita,Kyosuke
AU  - Li,Xiang
AU  - Huang,Zhe
AU  - Song,Haizhen
AU  - Asai,Masashi
AU  - Hao,Chang-Ning
AU  - Unno,Kazumasa
AU  - Koike,Teruhiro
AU  - Oshida,Yoshiharu
AU  - Okumura,Kenji
AU  - Murohara,Toyoaki
AU  - Kuzuya,Masafumi
TI  - Cathepsin K-mediated Notch1 activation contributes to neovascularization in response to hypoxia
SN  - 2041-1723
PY  - 2015///1001
KW  - Animals
KW  - Basic Helix-Loop-Helix Transcription Factors
KW  - metabolism
KW  - Capillaries
KW  - pathology
KW  - Cathepsin K
KW  - genetics
KW  - Cell Cycle Proteins
KW  - Femoral Artery
KW  - surgery
KW  - Hindlimb
KW  - blood supply
KW  - Homeodomain Proteins
KW  - Hypoxia
KW  - Ischemia
KW  - Laser-Doppler Flowmetry
KW  - Ligation
KW  - Mice
KW  - Mice, Knockout
KW  - Muscle, Skeletal
KW  - Neovascularization, Physiologic
KW  - Phosphorylation
KW  - Proto-Oncogene Proteins c-akt
KW  - RNA, Messenger
KW  - Receptor, Notch1
KW  - Repressor Proteins
KW  - Reverse Transcriptase Polymerase Chain Reaction
KW  - Transcription Factor HES-1
KW  - Vascular Endothelial Growth Factor A
KW  - Vascular Endothelial Growth Factor Receptor-1
N1  - Publication Type: Journal Article; Research Support, Non-U.S. Gov't
UR  - https://doi.org/10.1038/ncomms4838
ER  -