Wang, Hailong <br/><br/>
CtIP maintains stability at common fragile sites and inverted repeats by end resection-independent endonuclease activity.  [electronic resource] 

 -  Molecular cell  Jun 2014 
 - 1012-1021 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1097-4164 
<br/><br/><label>Standard No.: </label>10.1016/j.molcel.2014.04.012  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Acid Anhydride Hydrolases<br/>Alu Elements--genetics<br/>Base Composition--genetics<br/>Carrier Proteins--genetics<br/>Cell Cycle Proteins--genetics<br/>Cell Line<br/>Chromosome Fragile Sites--genetics<br/>DNA Breaks, Double-Stranded<br/>DNA Repair--genetics<br/>DNA Repair Enzymes--genetics<br/>DNA-Binding Proteins--genetics<br/>Endodeoxyribonucleases<br/>Endonucleases--genetics<br/>Homologous Recombination--genetics<br/>Humans<br/>Inverted Repeat Sequences--genetics<br/>MRE11 Homologue Protein<br/>Mitosis--genetics<br/>Nuclear Proteins--genetics<br/>Recombination, Genetic