Ranganathan, Punithavathi <br/><br/>
Semaphorin 3A inactivation suppresses ischemia-reperfusion-induced inflammation and acute kidney injury.  [electronic resource] 

 -  American journal of physiology. Renal physiology  Jul 2014 
 - F183-94 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1522-1466 
<br/><br/><label>Standard No.: </label>10.1152/ajprenal.00177.2014  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Acute Kidney Injury--genetics<br/>Animals<br/>Apoptosis<br/>Apoptosis Regulatory Proteins--metabolism<br/>Cell Line<br/>Cisplatin--toxicity<br/>Cytokines--metabolism<br/>Dendritic Cells--metabolism<br/>Disease Models, Animal<br/>Epithelial Cells--metabolism<br/>Inflammation Mediators--metabolism<br/>Kidney--drug effects<br/>Kidney Tubules--metabolism<br/>Macrophages--metabolism<br/>Mice<br/>Mice, Inbred C3H<br/>Mice, Inbred C57BL<br/>Mice, Knockout<br/>Mutation<br/>Nephritis--genetics<br/>Neuropilin-1--metabolism<br/>Reperfusion Injury--genetics<br/>Semaphorin-3A--antagonists & inhibitors<br/>Toll-Like Receptor 4--metabolism