Iwata, Naohiro <br/><br/>
Orally administrated ascorbic acid suppresses neuronal damage and modifies expression of SVCT2 and GLUT1 in the brain of diabetic rats with cerebral ischemia-reperfusion.  [electronic resource] 

 -  Nutrients  Apr 2014 
 - 1554-77 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>2072-6643 
<br/><br/><label>Standard No.: </label>10.3390/nu6041554  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Administration, Oral<br/>Animals<br/>Apoptosis--drug effects<br/>Ascorbic Acid--administration & dosage<br/>Brain--drug effects<br/>Caspase 3<br/>Diabetes Mellitus, Experimental--drug therapy<br/>Dietary Supplements<br/>Endothelial Cells--drug effects<br/>Glucose Transporter Type 1--genetics<br/>Infarction, Middle Cerebral Artery--drug therapy<br/>Interleukin-1beta--metabolism<br/>Male<br/>Neurons--drug effects<br/>Oxidative Stress--drug effects<br/>Rats<br/>Rats, Sprague-Dawley<br/>Sodium-Coupled Vitamin C Transporters--genetics<br/>Streptozocin<br/>Tumor Necrosis Factor-alpha--metabolism<br/>Up-Regulation