Hartung, Jane E <br/><br/>
β2- and β3-adrenergic receptors drive COMT-dependent pain by increasing production of nitric oxide and cytokines.  [electronic resource] 

 -  Pain  Jul 2014 
 - 1346-1355 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, N.I.H., Extramural 
<br/><br/><label>ISSN: </label>1872-6623 
<br/><br/><label>Standard No.: </label>10.1016/j.pain.2014.04.011  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Adrenergic beta-2 Receptor Antagonists--pharmacology<br/>Adrenergic beta-3 Receptor Antagonists--pharmacology<br/>Animals<br/>Catechol O-Methyltransferase--metabolism<br/>Catechol O-Methyltransferase Inhibitors--pharmacology<br/>Chemokine CCL2--drug effects<br/>Cytokines--metabolism<br/>Hyperalgesia--metabolism<br/>Interleukin-1beta--drug effects<br/>Interleukin-6--metabolism<br/>Male<br/>Nitric Oxide--metabolism<br/>Nitric Oxide Synthase--antagonists & inhibitors<br/>Rats<br/>Rats, Sprague-Dawley<br/>Receptors, Adrenergic, beta-2--metabolism<br/>Receptors, Adrenergic, beta-3--metabolism<br/>Tumor Necrosis Factor-alpha--drug effects