Huang, Jau-Shyang <br/><br/>
Klotho attenuates high glucose-induced fibronectin and cell hypertrophy via the ERK1/2-p38 kinase signaling pathway in renal interstitial fibroblasts.  [electronic resource] 

 -  Molecular and cellular endocrinology  Jun 2014 
 - 45-53 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1872-8057 
<br/><br/><label>Standard No.: </label>10.1016/j.mce.2014.04.001  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Cell Line<br/>Collagen--biosynthesis<br/>Diabetes Mellitus, Experimental--complications<br/>Diabetic Nephropathies--metabolism<br/>Fibroblasts--metabolism<br/>Fibronectins--metabolism<br/>Gene Expression<br/>Glucose--physiology<br/>Glucuronidase--physiology<br/>Hypertrophy--metabolism<br/>Kidney Cortex--metabolism<br/>Klotho Proteins<br/>MAP Kinase Signaling System<br/>Male<br/>Protein Serine-Threonine Kinases--metabolism<br/>Rats, Sprague-Dawley<br/>Receptor, Transforming Growth Factor-beta Type II<br/>Receptors, Transforming Growth Factor beta--metabolism<br/>Smad2 Protein--metabolism<br/>Smad3 Protein--metabolism<br/>Transforming Growth Factor beta--physiology<br/>p38 Mitogen-Activated Protein Kinases--metabolism