Flex, Elisabetta <br/><br/>
Activating mutations in RRAS underlie a phenotype within the RASopathy spectrum and contribute to leukaemogenesis.  [electronic resource] 

 -  Human molecular genetics  Aug 2014 
 - 4315-27 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1460-2083 
<br/><br/><label>Standard No.: </label>10.1093/hmg/ddu148  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Caenorhabditis elegans<br/>Carcinogenesis--genetics<br/>Cohort Studies<br/>Extracellular Signal-Regulated MAP Kinases--metabolism<br/>Humans<br/>Leukemia, Myeloid, Acute--genetics<br/>Leukemia, Myelomonocytic, Juvenile--genetics<br/>MAP Kinase Kinase Kinases--metabolism<br/>Mutation--physiology<br/>Noonan Syndrome--genetics<br/>Oncogene Protein v-akt--metabolism<br/>Phenotype<br/>Signal Transduction--genetics<br/>ras Proteins--chemistry