Ikeda, Aki <br/><br/>
Progression of autoimmune hepatitis is mediated by IL-18-producing dendritic cells and hepatic CXCL9 expression in mice.  [electronic resource] 

 -  Hepatology (Baltimore, Md.)  Jul 2014 
 - 224-36 p.  digital 
<br/><br/> Publication Type: Journal Article 
<br/><br/><label>ISSN: </label>1527-3350 
<br/><br/><label>Standard No.: </label>10.1002/hep.27087  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>CD8-Positive T-Lymphocytes--immunology<br/>Cell Differentiation--immunology<br/>Chemokine CXCL9--immunology<br/>Dendritic Cells--immunology<br/>Disease Models, Animal<br/>Female<br/>Hepatitis, Autoimmune--immunology<br/>Interferon-gamma--immunology<br/>Interleukin-18--immunology<br/>Liver--immunology<br/>Liver Failure, Acute--immunology<br/>Macrophages--immunology<br/>Male<br/>Mice<br/>Mice, Inbred BALB C<br/>Mice, Knockout<br/>Programmed Cell Death 1 Receptor--genetics<br/>Receptors, CXCR3--immunology<br/>Signal Transduction--immunology<br/>T-Box Domain Proteins--immunology<br/>Thymectomy<br/>T-bet Transcription Factor