Tong, Xiaoping <br/><br/>
Astrocyte Kir4.1 ion channel deficits contribute to neuronal dysfunction in Huntington's disease model mice.  [electronic resource] 

 -  Nature neuroscience  May 2014 
 - 694-703 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1546-1726 
<br/><br/><label>Standard No.: </label>10.1038/nn.3691  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Age Factors<br/>Animals<br/>Astrocytes--metabolism<br/>Corpus Striatum--physiopathology<br/>Disease Models, Animal<br/>Green Fluorescent Proteins--genetics<br/>HEK293 Cells<br/>Hindlimb Suspension--physiology<br/>Humans<br/>Huntingtin Protein<br/>Huntington Disease--genetics<br/>In Vitro Techniques<br/>Locomotion--genetics<br/>Mice<br/>Mice, Inbred C57BL<br/>Mice, Transgenic<br/>Nerve Tissue Proteins--genetics<br/>Neurons--metabolism<br/>Nuclear Proteins--genetics<br/>Potassium Channels, Inwardly Rectifying--deficiency<br/>Survival Analysis<br/>Trinucleotide Repeats--genetics<br/>Kcnj10 Channel