Ward, Jordan D <br/><br/>
Defects in the C. elegans acyl-CoA synthase, acs-3, and nuclear hormone receptor, nhr-25, cause sensitivity to distinct, but overlapping stresses.  [electronic resource] 

 -  PloS one  2014 
 - e92552 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, Non-P.H.S. 
<br/><br/><label>ISSN: </label>1932-6203 
<br/><br/><label>Standard No.: </label>10.1371/journal.pone.0092552  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Animals, Genetically Modified<br/>Antimicrobial Cationic Peptides--genetics<br/>Caenorhabditis elegans--genetics<br/>Coenzyme A Ligases--deficiency<br/>DNA-Binding Proteins--deficiency<br/>Gene Expression Regulation<br/>Gene Knockout Techniques<br/>Genetic Association Studies<br/>Longevity--genetics<br/>Mutation<br/>Phenotype<br/>RNA Interference<br/>Stress, Physiological<br/>Transcription Factors--deficiency<br/>Transcriptome<br/>Long-Chain-Fatty-Acid-CoA Ligase