Zhao, H <br/><br/>
Activation of glucagon-like peptide-1 receptor inhibits tumourigenicity and metastasis of human pancreatic cancer cells via PI3K/Akt pathway.  [electronic resource] 

 -  Diabetes, obesity & metabolism  Sep 2014 
 - 850-60 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1463-1326 
<br/><br/><label>Standard No.: </label>10.1111/dom.12291  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Androstadienes--pharmacology<br/>Animals<br/>Cell Line, Tumor<br/>Cell Movement--drug effects<br/>Cell Proliferation--drug effects<br/>Chromones--pharmacology<br/>Diabetes Mellitus, Type 2--complications<br/>Enzyme Inhibitors--pharmacology<br/>Gene Expression Regulation, Neoplastic--drug effects<br/>Glucagon-Like Peptide-1 Receptor--drug effects<br/>Humans<br/>Hypoglycemic Agents--pharmacology<br/>Immunohistochemistry<br/>Liraglutide--pharmacology<br/>Male<br/>Mice<br/>Mice, Nude<br/>Morpholines--pharmacology<br/>Neoplasm Invasiveness<br/>Pancreatic Neoplasms--etiology<br/>Phosphatidylinositol 3-Kinases--metabolism<br/>Phosphoinositide-3 Kinase Inhibitors<br/>Phosphorylation<br/>Proto-Oncogene Proteins c-akt--drug effects<br/>Signal Transduction--drug effects<br/>Wortmannin<br/>Xenograft Model Antitumor Assays