Moss, Nicholas G <br/><br/>
Renal vasoconstriction by vasopressin V1a receptors is modulated by nitric oxide, prostanoids, and superoxide but not the ADP ribosyl cyclase CD38.  [electronic resource] 

 -  American journal of physiology. Renal physiology  May 2014 
 - F1143-54 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, N.I.H., Extramural 
<br/><br/><label>ISSN: </label>1522-1466 
<br/><br/><label>Standard No.: </label>10.1152/ajprenal.00664.2013  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>ADP-ribosyl Cyclase 1--deficiency<br/>Animals<br/>Arginine Vasopressin--pharmacology<br/>Cyclic N-Oxides--pharmacology<br/>Dose-Response Relationship, Drug<br/>Kidney--blood supply<br/>Mice<br/>Mice, Inbred C57BL<br/>Mice, Knockout<br/>Models, Animal<br/>Nitric Oxide--physiology<br/>Prostaglandins--physiology<br/>Reactive Oxygen Species--metabolism<br/>Receptors, Vasopressin--drug effects<br/>Signal Transduction--drug effects<br/>Spin Labels<br/>Superoxides--metabolism<br/>Vasoconstriction--drug effects