Tang, Youcai <br/><br/>
Curcumin eliminates the effect of advanced glycation end-products (AGEs) on the divergent regulation of gene expression of receptors of AGEs by interrupting leptin signaling.  [electronic resource] 

 -  Laboratory investigation; a journal of technical methods and pathology  May 2014 
 - 503-16 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1530-0307 
<br/><br/><label>Standard No.: </label>10.1038/labinvest.2014.42  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Antineoplastic Agents--pharmacology<br/>Cells, Cultured<br/>Chromones<br/>Curcuma<br/>Curcumin--pharmacology<br/>Gene Expression Regulation--drug effects<br/>Glycation End Products, Advanced--antagonists & inhibitors<br/>Hepatic Stellate Cells--drug effects<br/>Leptin--antagonists & inhibitors<br/>Liver Cirrhosis--prevention & control<br/>Male<br/>Mice<br/>Mice, Inbred C57BL<br/>Morpholines<br/>Plant Extracts--pharmacology<br/>Rats<br/>Rats, Sprague-Dawley<br/>Receptor for Advanced Glycation End Products<br/>Receptors, Immunologic--metabolism<br/>Receptors, Leptin--metabolism<br/>Tyrphostins