Huang, Jie <br/><br/>
Genome-wide association study for circulating tissue plasminogen activator levels and functional follow-up implicates endothelial STXBP5 and STX2.  [electronic resource] 

 -  Arteriosclerosis, thrombosis, and vascular biology  May 2014 
 - 1093-101 p.  digital 
<br/><br/> Publication Type: Journal Article; Meta-Analysis; Research Support, N.I.H., Extramural 
<br/><br/><label>ISSN: </label>1524-4636 
<br/><br/><label>Standard No.: </label>10.1161/ATVBAHA.113.302088  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Aged<br/>Cells, Cultured<br/>Coronary Artery Disease--blood<br/>Endothelial Cells--enzymology<br/>Europe<br/>Female<br/>Gene Expression Regulation<br/>Gene Silencing<br/>Genetic Loci<br/>Genetic Predisposition to Disease<br/>Genome-Wide Association Study<br/>Humans<br/>Male<br/>Middle Aged<br/>Nerve Tissue Proteins--genetics<br/>Phenotype<br/>Polymorphism, Single Nucleotide<br/>R-SNARE Proteins--genetics<br/>Risk Factors<br/>Stroke--blood<br/>Syntaxin 1--genetics<br/>Tissue Plasminogen Activator--blood<br/>Transfection<br/>United States<br/>Up-Regulation