Lemarchant, Sighild <br/><br/>
tPA promotes ADAMTS-4-induced CSPG degradation, thereby enhancing neuroplasticity following spinal cord injury.  [electronic resource] 

 -  Neurobiology of disease  Jun 2014 
 - 28-42 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1095-953X 
<br/><br/><label>Standard No.: </label>10.1016/j.nbd.2014.02.005  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>ADAM Proteins--metabolism<br/>ADAMTS4 Protein<br/>Animals<br/>Axons--drug effects<br/>Cells, Cultured<br/>Chondroitin Sulfate Proteoglycans--metabolism<br/>Female<br/>Neurites--drug effects<br/>Neurocan<br/>Neuronal Plasticity--drug effects<br/>Neuropeptides--pharmacology<br/>Neuroprotective Agents--pharmacology<br/>Plasminogen Activator Inhibitor 1--pharmacology<br/>Procollagen N-Endopeptidase--metabolism<br/>Rats<br/>Rats, Wistar<br/>Recovery of Function<br/>Serine Proteinase Inhibitors--pharmacology<br/>Serpins--pharmacology<br/>Spinal Cord--drug effects<br/>Spinal Cord Compression--drug therapy<br/>Spinal Cord Injuries--drug therapy<br/>Tissue Plasminogen Activator--antagonists & inhibitors<br/>Neuroserpin