Carlson, Thaddeus J <br/><br/>
Halofuginone-induced amino acid starvation regulates Stat3-dependent Th17 effector function and reduces established autoimmune inflammation.  [electronic resource] 

 -  Journal of immunology (Baltimore, Md. : 1950)  Mar 2014 
 - 2167-76 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1550-6606 
<br/><br/><label>Standard No.: </label>10.4049/jimmunol.1302316  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Amino Acids--deficiency<br/>Animals<br/>Encephalomyelitis, Autoimmune, Experimental--genetics<br/>Inflammation--chemically induced<br/>Interleukin-23--genetics<br/>Lymphocyte Activation<br/>Mice<br/>Mice, Inbred BALB C<br/>Mice, Knockout<br/>Phosphorylation--genetics<br/>Piperidines<br/>Quinazolinones<br/>Receptors, CCR6--genetics<br/>Receptors, Interleukin--genetics<br/>STAT3 Transcription Factor--genetics<br/>Th17 Cells--immunology