Rieber, Manuel <br/><br/>
p53 inactivation decreases dependence on estrogen/ERK signalling for proliferation but promotes EMT and susceptility to 3-bromopyruvate in ERα+ breast cancer MCF-7 cells.  [electronic resource] 

 -  Biochemical pharmacology  Mar 2014 
 - 169-77 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1873-2968 
<br/><br/><label>Standard No.: </label>10.1016/j.bcp.2014.01.025  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Breast Neoplasms--drug therapy<br/>Cell Proliferation--drug effects<br/>Epithelial-Mesenchymal Transition--drug effects<br/>Estrogen Receptor alpha--biosynthesis<br/>Female<br/>Genes, p53--drug effects<br/>Humans<br/>MAP Kinase Signaling System--drug effects<br/>MCF-7 Cells<br/>Pyruvate Dehydrogenase Complex--antagonists & inhibitors<br/>Pyruvates--pharmacology