Wang, JuFang <br/><br/>
Induced overexpression of phospholemman S68E mutant improves cardiac contractility and mortality after ischemia-reperfusion.  [electronic resource] 

 -  American journal of physiology. Heart and circulatory physiology  Apr 2014 
 - H1066-77 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, N.I.H., Extramural 
<br/><br/><label>ISSN: </label>1522-1539 
<br/><br/><label>Standard No.: </label>10.1152/ajpheart.00861.2013  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Adrenergic beta-Agonists--pharmacology<br/>Animals<br/>Calcium Channels, L-Type--metabolism<br/>Calcium Signaling<br/>Cardiotonic Agents--pharmacology<br/>Disease Models, Animal<br/>Male<br/>Membrane Proteins--genetics<br/>Mice<br/>Mice, Transgenic<br/>Mutation<br/>Myocardial Contraction--drug effects<br/>Myocardial Infarction--drug therapy<br/>Myocardial Reperfusion Injury--drug therapy<br/>Phosphoproteins--genetics<br/>Recovery of Function<br/>Ryanodine Receptor Calcium Release Channel--metabolism<br/>Sarcoplasmic Reticulum--metabolism<br/>Sarcoplasmic Reticulum Calcium-Transporting ATPases--metabolism<br/>Sodium-Calcium Exchanger--metabolism<br/>Sodium-Potassium-Exchanging ATPase--metabolism<br/>Stroke Volume<br/>Up-Regulation<br/>Ventricular Dysfunction, Left--metabolism<br/>Ventricular Function, Left--drug effects<br/>Ventricular Pressure