Heard, Melissa E <br/><br/>
Krüppel-like factor 9 deficiency in uterine endometrial cells promotes ectopic lesion establishment associated with activated notch and hedgehog signaling in a mouse model of endometriosis.  [electronic resource] 

 -  Endocrinology  Apr 2014 
 - 1532-46 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1945-7170 
<br/><br/><label>Standard No.: </label>10.1210/en.2013-1947  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Apoptosis<br/>Ascitic Fluid--metabolism<br/>Cell Proliferation<br/>Disease Models, Animal<br/>Endometriosis--genetics<br/>Endometrium--cytology<br/>Female<br/>Gene Expression Regulation<br/>Hedgehog Proteins--metabolism<br/>Kruppel-Like Transcription Factors--genetics<br/>Mice<br/>Mice, Knockout<br/>Receptors, Estrogen--metabolism<br/>Receptors, Notch--metabolism<br/>Receptors, Steroid--metabolism<br/>Signal Transduction