Redente, Elizabeth F <br/><br/>
Tumor necrosis factor-α accelerates the resolution of established pulmonary fibrosis in mice by targeting profibrotic lung macrophages.  [electronic resource] 

 -  American journal of respiratory cell and molecular biology  Apr 2014 
 - 825-37 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1535-4989 
<br/><br/><label>Standard No.: </label>10.1165/rcmb.2013-0386OC  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Bleomycin<br/>Cells, Cultured<br/>Disease Models, Animal<br/>Hydroxyproline--metabolism<br/>Idiopathic Pulmonary Fibrosis--drug therapy<br/>Lung--drug effects<br/>Lung Compliance<br/>Macrophages, Alveolar--drug effects<br/>Male<br/>Mice<br/>Mice, Inbred C57BL<br/>Mice, Knockout<br/>Mice, Transgenic<br/>Receptor, Macrophage Colony-Stimulating Factor--genetics<br/>Recovery of Function<br/>Remission Induction<br/>Tacrolimus Binding Proteins--genetics<br/>Time Factors<br/>Tumor Necrosis Factor-alpha--deficiency<br/>fas Receptor--genetics