Dau, Alejandro <br/><br/>
Chronic blockade of extrasynaptic NMDA receptors ameliorates synaptic dysfunction and pro-death signaling in Huntington disease transgenic mice.  [electronic resource] 

 -  Neurobiology of disease  Feb 2014 
 - 533-42 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1095-953X 
<br/><br/><label>Standard No.: </label>10.1016/j.nbd.2013.11.013  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>CREB-Binding Protein--metabolism<br/>Calcium Signaling<br/>Calpain--metabolism<br/>Corpus Striatum--drug effects<br/>Excitatory Amino Acid Antagonists--pharmacology<br/>Huntington Disease--genetics<br/>Memantine--pharmacology<br/>Mice<br/>Mice, Transgenic<br/>Receptors, N-Methyl-D-Aspartate--antagonists & inhibitors<br/>Synapses--drug effects<br/>p38 Mitogen-Activated Protein Kinases--metabolism