Ma, Jiacheng <br/><br/>
Heat shock protein 70 is necessary to improve mitochondrial bioenergetics and reverse diabetic sensory neuropathy following KU-32 therapy.  [electronic resource] 

 -  The Journal of pharmacology and experimental therapeutics  Feb 2014 
 - 281-92 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1521-0103 
<br/><br/><label>Standard No.: </label>10.1124/jpet.113.210435  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Cells, Cultured<br/>Diabetes Mellitus, Type 1--complications<br/>Diabetes Mellitus, Type 2--complications<br/>Diabetic Neuropathies--prevention & control<br/>Dose-Response Relationship, Drug<br/>Female<br/>Ganglia, Spinal--drug effects<br/>HSP70 Heat-Shock Proteins--genetics<br/>Hypoglycemic Agents--administration & dosage<br/>Male<br/>Mice<br/>Mice, Inbred C57BL<br/>Mice, Knockout<br/>Mice, Transgenic<br/>Mitochondria--drug effects<br/>Mitochondrial Dynamics--drug effects<br/>Neuritis--prevention & control<br/>Neurons--drug effects<br/>Neuroprotective Agents--administration & dosage<br/>Novobiocin--administration & dosage<br/>Oxidative Phosphorylation--drug effects<br/>Sensory Receptor Cells--drug effects