Forrester, Alison R <br/><br/>
Induction of a chloracne phenotype in an epidermal equivalent model by 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD) is dependent on aryl hydrocarbon receptor activation and is not reproduced by aryl hydrocarbon receptor knock down.  [electronic resource] 

 -  Journal of dermatological science  Jan 2014 
 - 10-22 p.  digital 
<br/><br/> Publication Type: Comparative Study; Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1873-569X 
<br/><br/><label>Standard No.: </label>10.1016/j.jdermsci.2013.09.001  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Basic Helix-Loop-Helix Transcription Factors--agonists<br/>Cell Differentiation--drug effects<br/>Cells, Cultured<br/>Chloracne--etiology<br/>Cytochrome P-450 CYP1A1--biosynthesis<br/>Dose-Response Relationship, Drug<br/>Enzyme Induction<br/>Epidermis--drug effects<br/>Filaggrin Proteins<br/>Gene Knockdown Techniques<br/>Humans<br/>Indoles--toxicity<br/>Intermediate Filament Proteins--metabolism<br/>Keratinocytes--drug effects<br/>Ligands<br/>Phenotype<br/>Polychlorinated Dibenzodioxins--toxicity<br/>Protein Precursors--metabolism<br/>RNA Interference<br/>Receptors, Aryl Hydrocarbon--agonists<br/>Thiazoles--toxicity<br/>Transfection<br/>Transglutaminases--metabolism<br/>beta-Naphthoflavone--toxicity