Lam, C R I <br/><br/>
Loss of TAK1 increases cell traction force in a ROS-dependent manner to drive epithelial-mesenchymal transition of cancer cells.  [electronic resource] 

 -  Cell death & disease  Oct 2013 
 - e848 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>2041-4889 
<br/><br/><label>Standard No.: </label>10.1038/cddis.2013.339  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Biomechanical Phenomena--drug effects<br/>Cell Line, Tumor<br/>Cell Membrane--drug effects<br/>Epithelial-Mesenchymal Transition--drug effects<br/>HEK293 Cells<br/>Humans<br/>Integrin beta1--metabolism<br/>Integrin beta3--metabolism<br/>MAP Kinase Kinase Kinases--deficiency<br/>Mice<br/>Models, Biological<br/>Neoplasm Invasiveness<br/>Neoplasms, Squamous Cell--enzymology<br/>Oxidation-Reduction--drug effects<br/>Reactive Oxygen Species--metabolism<br/>Signal Transduction--drug effects<br/>Skin Neoplasms--enzymology<br/>Smad Proteins--metabolism<br/>Transforming Growth Factor beta1--pharmacology<br/>rac1 GTP-Binding Protein--metabolism<br/>rho GTP-Binding Proteins--metabolism