Hall, Bradford E <br/><br/>
Transforming growth factor-β3 (TGF-β3) knock-in ameliorates inflammation due to TGF-β1 deficiency while promoting glucose tolerance.  [electronic resource] 

 -  The Journal of biological chemistry  Nov 2013 
 - 32074-92 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, N.I.H., Intramural 
<br/><br/><label>ISSN: </label>1083-351X 
<br/><br/><label>Standard No.: </label>10.1074/jbc.M113.480764  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>COS Cells<br/>Chlorocebus aethiops<br/>Gene Knock-In Techniques<br/>Glucose--genetics<br/>Hep G2 Cells<br/>Humans<br/>Inflammation--genetics<br/>Mice<br/>Mice, Inbred BALB C<br/>Mice, Transgenic<br/>Neovascularization, Physiologic--physiology<br/>Protein Isoforms--genetics<br/>Recombinant Fusion Proteins--genetics<br/>Signal Transduction--physiology<br/>Swine<br/>Transforming Growth Factor beta1--genetics<br/>Transforming Growth Factor beta3--genetics