Liu, Yang <br/><br/>
Inactivation of Cdc42 in neural crest cells causes craniofacial and cardiovascular morphogenesis defects.  [electronic resource] 

 -  Developmental biology  Nov 2013 
 - 239-52 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1095-564X 
<br/><br/><label>Standard No.: </label>10.1016/j.ydbio.2013.09.013  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Actins--metabolism<br/>Animals<br/>Bone Morphogenetic Protein 2--pharmacology<br/>Cardiovascular Abnormalities--embryology<br/>Cell Differentiation--drug effects<br/>Cell Movement--drug effects<br/>Craniofacial Abnormalities--embryology<br/>Crosses, Genetic<br/>Cytoskeleton--metabolism<br/>Embryo, Mammalian--abnormalities<br/>Enzyme Activation--drug effects<br/>Female<br/>Gene Deletion<br/>Genotype<br/>Male<br/>Mice<br/>Mice, Knockout<br/>Morphogenesis--drug effects<br/>Neural Crest--drug effects<br/>Osteogenesis--drug effects<br/>Phenotype<br/>Pseudopodia--drug effects<br/>Thymus Gland--abnormalities<br/>cdc42 GTP-Binding Protein--metabolism