Abe, Yoshifusa <br/><br/>
TGF-β1 stimulates mitochondrial oxidative phosphorylation and generation of reactive oxygen species in cultured mouse podocytes, mediated in part by the mTOR pathway.  [electronic resource] 

 -  American journal of physiology. Renal physiology  Nov 2013 
 - F1477-90 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, N.I.H., Extramural 
<br/><br/><label>ISSN: </label>1522-1466 
<br/><br/><label>Standard No.: </label>10.1152/ajprenal.00182.2013  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Adenosine Triphosphate--metabolism<br/>Animals<br/>Antigens, Polyomavirus Transforming--genetics<br/>Apoptosis--drug effects<br/>Biosensing Techniques<br/>Cells, Cultured<br/>Dose-Response Relationship, Drug<br/>Fatty Acids--metabolism<br/>Glycolysis--drug effects<br/>Humans<br/>Hydrogen-Ion Concentration<br/>Intracellular Signaling Peptides and Proteins--genetics<br/>Membrane Potential, Mitochondrial--drug effects<br/>Membrane Proteins--genetics<br/>Mice<br/>Mice, Transgenic<br/>Mitochondria--drug effects<br/>Oxidative Phosphorylation--drug effects<br/>Podocytes--drug effects<br/>Protein Kinase Inhibitors--pharmacology<br/>Reactive Oxygen Species--metabolism<br/>Recombinant Proteins--pharmacology<br/>Signal Transduction--drug effects<br/>TOR Serine-Threonine Kinases--antagonists & inhibitors<br/>Time Factors<br/>Transforming Growth Factor beta1--pharmacology