Seow, Vernon <br/><br/>
Inflammatory responses induced by lipopolysaccharide are amplified in primary human monocytes but suppressed in macrophages by complement protein C5a.  [electronic resource] 

 -  Journal of immunology (Baltimore, Md. : 1950)  Oct 2013 
 - 4308-16 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1550-6606 
<br/><br/><label>Standard No.: </label>10.4049/jimmunol.1301355  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Aminoquinolines<br/>Butadienes--pharmacology<br/>Cells, Cultured<br/>Complement C5a--metabolism<br/>Extracellular Signal-Regulated MAP Kinases--antagonists & inhibitors<br/>GTP-Binding Protein alpha Subunits, Gi-Go--antagonists & inhibitors<br/>Granulocyte-Macrophage Colony-Stimulating Factor<br/>Humans<br/>Imiquimod<br/>Inflammation--chemically induced<br/>Interleukin-10--biosynthesis<br/>Interleukin-6--metabolism<br/>JNK Mitogen-Activated Protein Kinases--metabolism<br/>Lipopolysaccharides<br/>MAP Kinase Signaling System--immunology<br/>Macrophage Colony-Stimulating Factor<br/>Macrophages--metabolism<br/>Mitogen-Activated Protein Kinase Kinases--antagonists & inhibitors<br/>Monocytes--metabolism<br/>Nitriles--pharmacology<br/>Pertussis Toxin--pharmacology<br/>Phosphatidylinositol 3-Kinases--metabolism<br/>Poly I-C<br/>Proto-Oncogene Proteins c-raf--metabolism<br/>Receptor, Anaphylatoxin C5a<br/>Receptors, Complement--metabolism<br/>Salmonella typhimurium--immunology<br/>Toll-Like Receptor 4--metabolism<br/>Tumor Necrosis Factors--metabolism