He, Kang

Lipopolysaccharide-induced cross-tolerance against renal ischemia-reperfusion injury is mediated by hypoxia-inducible factor-2α-regulated nitric oxide production. [electronic resource] - Kidney international Feb 2014 - 276-88 p. digital

Publication Type: Journal Article; Research Support, Non-U.S. Gov't

1523-1755

10.1038/ki.2013.342 doi


Acute Kidney Injury--genetics
Animals
Basic Helix-Loop-Helix Transcription Factors--deficiency
Bone Marrow Transplantation
Cells, Cultured
Cytoprotection
Disease Models, Animal
Endothelial Cells--drug effects
Enzyme Inhibitors--pharmacology
Human Umbilical Vein Endothelial Cells--drug effects
Humans
Hypoxia-Inducible Factor 1, alpha Subunit--genetics
Kidney--blood supply
Lipopolysaccharides--pharmacology
Male
Mice
Mice, Knockout
Microcirculation--drug effects
NF-kappa B--metabolism
Nitric Oxide--metabolism
Nitric Oxide Donors--pharmacology
Nitric Oxide Synthase Type II--antagonists & inhibitors
Nitric Oxide Synthase Type III--antagonists & inhibitors
RNA Interference
Renal Circulation--drug effects
Reperfusion Injury--genetics
Signal Transduction
Time Factors
Transfection
Warm Ischemia