Azevedo, E P <br/><br/>
Activated microglia mediate synapse loss and short-term memory deficits in a mouse model of transthyretin-related oculoleptomeningeal amyloidosis.  [electronic resource] 

 -  Cell death & disease  Sep 2013 
 - e789 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>2041-4889 
<br/><br/><label>Standard No.: </label>10.1038/cddis.2013.325  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Amyloid<br/>Amyloid Neuropathies, Familial--complications<br/>Animals<br/>Brain--metabolism<br/>Cell Death--drug effects<br/>Cell Nucleus--metabolism<br/>Cells, Cultured<br/>Culture Media, Conditioned--pharmacology<br/>Disease Models, Animal<br/>Endocytosis<br/>Glycogen Synthase Kinase 3--metabolism<br/>Glycogen Synthase Kinase 3 beta<br/>Inflammation Mediators--metabolism<br/>Interleukin-6--metabolism<br/>Memory Disorders--complications<br/>Memory, Short-Term--drug effects<br/>Mice<br/>Microglia--drug effects<br/>Minocycline--pharmacology<br/>Mutation--genetics<br/>NF-kappa B--metabolism<br/>Neurons--drug effects<br/>Phosphorylation<br/>Prealbumin--metabolism<br/>Protein Transport<br/>Proto-Oncogene Proteins c-akt--metabolism<br/>Synapses--drug effects<br/>Tumor Necrosis Factor-alpha--metabolism