Janes, Kali <br/><br/>
Bioenergetic deficits in peripheral nerve sensory axons during chemotherapy-induced neuropathic pain resulting from peroxynitrite-mediated post-translational nitration of mitochondrial superoxide dismutase.  [electronic resource] 

 -  Pain  Nov 2013 
 - 2432-2440 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1872-6623 
<br/><br/><label>Standard No.: </label>10.1016/j.pain.2013.07.032  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Adenosine Triphosphate--metabolism<br/>Animals<br/>Antineoplastic Agents--adverse effects<br/>Antineoplastic Agents, Phytogenic--pharmacology<br/>Axons--physiology<br/>Boronic Acids--pharmacology<br/>Bortezomib<br/>Energy Metabolism--physiology<br/>Hyperalgesia--drug therapy<br/>Male<br/>Mitochondria--drug effects<br/>Neoplasm Transplantation<br/>Neuralgia--chemically induced<br/>Organoplatinum Compounds--pharmacology<br/>Oxaliplatin<br/>Paclitaxel--pharmacology<br/>Peripheral Nerves--physiology<br/>Peroxynitrous Acid--physiology<br/>Physical Stimulation<br/>Protein Processing, Post-Translational--physiology<br/>Pyrazines--pharmacology<br/>Rats<br/>Rats, Sprague-Dawley<br/>Sensory Receptor Cells--physiology<br/>Superoxide Dismutase--metabolism