Chen, Wen <br/><br/>
Atrial natriuretic peptide-mediated inhibition of microcirculatory endothelial Ca2+ and permeability response to histamine involves cGMP-dependent protein kinase I and TRPC6 channels.  [electronic resource] 

 -  Arteriosclerosis, thrombosis, and vascular biology  Sep 2013 
 - 2121-9 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1524-4636 
<br/><br/><label>Standard No.: </label>10.1161/ATVBAHA.113.001974  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Anti-Inflammatory Agents--pharmacology<br/>Atrial Natriuretic Factor--pharmacology<br/>Calcium--metabolism<br/>Calcium Channel Blockers--pharmacology<br/>Capillary Permeability--drug effects<br/>Cyclic GMP-Dependent Protein Kinase Type I--deficiency<br/>Dose-Response Relationship, Drug<br/>Endothelial Cells--drug effects<br/>HEK293 Cells<br/>Histamine--pharmacology<br/>Histamine Antagonists--pharmacology<br/>Human Umbilical Vein Endothelial Cells--drug effects<br/>Humans<br/>Male<br/>Mast Cells--drug effects<br/>Membrane Proteins<br/>Mice<br/>Mice, Knockout<br/>Microvessels--drug effects<br/>Phosphodiesterase 5 Inhibitors--pharmacology<br/>Phosphoproteins--genetics<br/>Phosphorylation<br/>Receptors, Atrial Natriuretic Factor--genetics<br/>Signal Transduction--drug effects<br/>TRPC Cation Channels--deficiency<br/>TRPC6 Cation Channel<br/>Time Factors<br/>Transfection