Rajendran, Praveen <br/><br/>
HDAC turnover, CtIP acetylation and dysregulated DNA damage signaling in colon cancer cells treated with sulforaphane and related dietary isothiocyanates.  [electronic resource] 

 -  Epigenetics  Jun 2013 
 - 612-23 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, N.I.H., Extramural 
<br/><br/><label>ISSN: </label>1559-2308 
<br/><br/><label>Standard No.: </label>10.4161/epi.24710  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Acetylation<br/>Antineoplastic Agents--pharmacology<br/>Apoptosis<br/>Autophagy<br/>Carrier Proteins--metabolism<br/>Cell Cycle Checkpoints<br/>Cell Line<br/>Cell Line, Tumor<br/>Colon--cytology<br/>Colonic Neoplasms--genetics<br/>DNA Damage<br/>Endodeoxyribonucleases<br/>Gene Expression<br/>Histone Deacetylase Inhibitors--pharmacology<br/>Histone Deacetylases--genetics<br/>Humans<br/>Isothiocyanates--administration & dosage<br/>Nuclear Proteins--metabolism<br/>Sulfoxides