Reelin, an extracellular matrix protein linked to early onset psychiatric diseases, drives postnatal development of the prefrontal cortex via GluN2B-NMDARs and the mTOR pathway. [electronic resource]
- Molecular psychiatry Apr 2014
- 417-26 p. digital
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
1476-5578
10.1038/mp.2013.66 doi
Analysis of Variance Animals Animals, Newborn Cell Adhesion Molecules, Neuronal--deficiency Conditioning, Classical--drug effects Dendritic Spines--drug effects Dose-Response Relationship, Drug Excitatory Amino Acid Agents--pharmacology Extinction, Psychological--drug effects Extracellular Matrix Proteins--deficiency Fear--drug effects Female In Vitro Techniques Ketamine--pharmacology Long-Term Potentiation--drug effects Male Mice Mice, Inbred C57BL Mice, Knockout Nerve Tissue Proteins--deficiency Patch-Clamp Techniques Phenols Piperidines--pharmacology Prefrontal Cortex--cytology Receptors, N-Methyl-D-Aspartate--metabolism Reelin Protein Serine Endopeptidases--deficiency Signal Transduction--drug effects Sirolimus TOR Serine-Threonine Kinases--metabolism