Freches, Danielle <br/><br/>
Mice genetically inactivated in interleukin-17A receptor are defective in long-term control of Mycobacterium tuberculosis infection.  [electronic resource] 

 -  Immunology  Oct 2013 
 - 220-31 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1365-2567 
<br/><br/><label>Standard No.: </label>10.1111/imm.12130  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Cytokines--immunology<br/>Enzyme-Linked Immunosorbent Assay<br/>Flow Cytometry<br/>Mice<br/>Mice, Inbred C57BL<br/>Mice, Knockout<br/>Mycobacterium tuberculosis<br/>Receptors, Interleukin-17--deficiency<br/>Tuberculosis--immunology