Wu, Minyan <br/><br/>
The anticancer effect of cytotoxin 1 from Naja atra Cantor venom is mediated by a lysosomal cell death pathway involving lysosomal membrane permeabilization and cathepsin B release.  [electronic resource] 

 -  The American journal of Chinese medicine  2013 
 - 643-63 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1793-6853 
<br/><br/><label>Standard No.: </label>10.1142/S0192415X13500456  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Antineoplastic Agents--pharmacology<br/>Apoptosis--drug effects<br/>Cathepsin B--metabolism<br/>Cell Line, Tumor<br/>Cell Survival--drug effects<br/>Cyclophosphamide--pharmacology<br/>Dose-Response Relationship, Drug<br/>Elapid Venoms--chemistry<br/>Elapidae<br/>Humans<br/>Intracellular Membranes--drug effects<br/>K562 Cells<br/>Leukemia P388--drug therapy<br/>Lysosomes--drug effects<br/>MCF-7 Cells<br/>Male<br/>Membrane Potential, Mitochondrial--drug effects<br/>Mice<br/>Mice, Inbred Strains<br/>Mitochondria--drug effects<br/>Neoplasms--drug therapy<br/>Permeability