Nayak, Kasturi Bala <br/><br/>
EVI1 targets ΔNp63 and upregulates the cyclin dependent kinase inhibitor p21 independent of p53 to delay cell cycle progression and cell proliferation in colon cancer cells.  [electronic resource] 

 -  The international journal of biochemistry & cell biology  Aug 2013 
 - 1568-76 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1878-5875 
<br/><br/><label>Standard No.: </label>10.1016/j.biocel.2013.04.032  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Base Sequence<br/>Cell Cycle<br/>Cell Proliferation<br/>Colonic Neoplasms--genetics<br/>Cyclin-Dependent Kinase Inhibitor p21--genetics<br/>DNA-Binding Proteins--chemistry<br/>Down-Regulation--genetics<br/>Gene Expression Regulation, Neoplastic<br/>Gene Silencing<br/>HCT116 Cells<br/>HEK293 Cells<br/>HT29 Cells<br/>Humans<br/>MDS1 and EVI1 Complex Locus Protein<br/>Molecular Sequence Data<br/>Promoter Regions, Genetic--genetics<br/>Protein Binding--genetics<br/>Protein Structure, Tertiary<br/>Proto-Oncogene Mas<br/>Proto-Oncogenes<br/>Transcription Factors--chemistry<br/>Transcription, Genetic<br/>Tumor Suppressor Protein p53--deficiency<br/>Tumor Suppressor Proteins--metabolism<br/>Up-Regulation--genetics<br/>Zinc Fingers