Varadarajan, Shankar <br/><br/>
Sabutoclax (BI97C1) and BI112D1, putative inhibitors of MCL-1, induce mitochondrial fragmentation either upstream of or independent of apoptosis.  [electronic resource] 

 -  Neoplasia (New York, N.Y.)  May 2013 
 - 568-78 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1476-5586 
<br/><br/><label>Standard No.: </label>10.1593/neo.13230  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Adenosine Triphosphate--metabolism<br/>Aniline Compounds--pharmacology<br/>Animals<br/>Antineoplastic Agents--pharmacology<br/>Apoptosis--drug effects<br/>Cell Line<br/>Dynamins<br/>GTP Phosphohydrolases--metabolism<br/>Gossypol--analogs & derivatives<br/>Humans<br/>Membrane Potential, Mitochondrial--drug effects<br/>Mice<br/>Microtubule-Associated Proteins--metabolism<br/>Mitochondria--drug effects<br/>Mitochondrial Dynamics--drug effects<br/>Mitochondrial Proteins--metabolism<br/>Myeloid Cell Leukemia Sequence 1 Protein--antagonists & inhibitors<br/>Reactive Oxygen Species--metabolism<br/>Sulfonamides--pharmacology