Coldewey, Sina M <br/><br/>
Erythropoietin attenuates acute kidney dysfunction in murine experimental sepsis by activation of the β-common receptor.  [electronic resource] 

 -  Kidney international  Sep 2013 
 - 482-90 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1523-1755 
<br/><br/><label>Standard No.: </label>10.1038/ki.2013.118  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Acute Kidney Injury--metabolism<br/>Animals<br/>Caspase 3--metabolism<br/>Cecum--physiopathology<br/>Cytokine Receptor Common beta Subunit--deficiency<br/>Disease Models, Animal<br/>Erythropoietin--pharmacology<br/>Glycogen Synthase Kinase 3--metabolism<br/>Glycogen Synthase Kinase 3 beta<br/>Hepatitis A Virus Cellular Receptor 1<br/>Kidney--drug effects<br/>Ligation<br/>Lipopolysaccharides--adverse effects<br/>Male<br/>Membrane Proteins--metabolism<br/>Mice<br/>Mice, Inbred C57BL<br/>Mice, Knockout<br/>Phosphorylation--drug effects<br/>Proto-Oncogene Proteins c-akt--metabolism<br/>Sepsis--chemically induced<br/>Signal Transduction--drug effects