Bojalil, Rafael <br/><br/>
Anti-tumor necrosis factor VNAR single domains reduce lethality and regulate underlying inflammatory response in a murine model of endotoxic shock.  [electronic resource] 

 -  BMC immunology  Apr 2013 
 - 17 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1471-2172 
<br/><br/><label>Standard No.: </label>10.1186/1471-2172-14-17  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Biomarkers--blood<br/>Disease Models, Animal<br/>Humans<br/>Inflammation--blood<br/>Interleukin-10--metabolism<br/>Interleukin-6--metabolism<br/>Lipopolysaccharides<br/>Liver--metabolism<br/>Male<br/>Mice<br/>Mice, Inbred BALB C<br/>Nitrates--metabolism<br/>Nitric Oxide Synthase Type II--metabolism<br/>Protein Structure, Tertiary<br/>RNA, Messenger--genetics<br/>Shock, Septic--blood<br/>Single-Domain Antibodies--therapeutic use<br/>Survival Analysis<br/>Treatment Outcome<br/>Tumor Necrosis Factor-alpha--immunology