Ma, Lei <br/><br/>
Cancer stem-like cell properties are regulated by EGFR/AKT/β-catenin signaling and preferentially inhibited by gefitinib in nasopharyngeal carcinoma.  [electronic resource] 

 -  The FEBS journal  May 2013 
 - 2027-41 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1742-4658 
<br/><br/><label>Standard No.: </label>10.1111/febs.12226  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Antineoplastic Agents--pharmacology<br/>Carcinoma--drug therapy<br/>Cell Line, Tumor<br/>Cell Proliferation<br/>Cell Survival--drug effects<br/>Cell Transformation, Neoplastic--metabolism<br/>Cisplatin--pharmacology<br/>ErbB Receptors--antagonists & inhibitors<br/>Gefitinib<br/>Gene Knockdown Techniques<br/>Homeodomain Proteins--metabolism<br/>Humans<br/>Mice<br/>Mice, Inbred BALB C<br/>Mice, Nude<br/>Nanog Homeobox Protein<br/>Nasopharyngeal Carcinoma<br/>Nasopharyngeal Neoplasms--drug therapy<br/>Neoplasm Transplantation<br/>Neoplastic Stem Cells--drug effects<br/>Proto-Oncogene Proteins c-akt--metabolism<br/>Quinazolines--pharmacology<br/>Side-Population Cells--drug effects<br/>Signal Transduction<br/>Spheroids, Cellular--metabolism<br/>Xenograft Model Antitumor Assays<br/>beta Catenin--genetics