Qing, Yan <br/><br/>
Apoptosis induced by trimethyltin chloride in human neuroblastoma cells SY5Y is regulated by a balance and cross-talk between NF-κB and MAPKs signaling pathways.  [electronic resource] 

 -  Archives of toxicology  Jul 2013 
 - 1273-85 p.  digital 
<br/><br/> Publication Type: Journal Article 
<br/><br/><label>ISSN: </label>1432-0738 
<br/><br/><label>Standard No.: </label>10.1007/s00204-013-1021-9  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Apoptosis--drug effects<br/>Cell Line, Tumor<br/>Cell Survival--drug effects<br/>Dose-Response Relationship, Drug<br/>Enzyme Activation<br/>Extracellular Signal-Regulated MAP Kinases--metabolism<br/>Humans<br/>I-kappa B Proteins--metabolism<br/>JNK Mitogen-Activated Protein Kinases--metabolism<br/>MAP Kinase Signaling System--drug effects<br/>NF-KappaB Inhibitor alpha<br/>NF-kappa B--metabolism<br/>Neuroblastoma--enzymology<br/>Neurons--drug effects<br/>Phosphorylation<br/>Proteasome Endopeptidase Complex--metabolism<br/>Proteasome Inhibitors--pharmacology<br/>Protein Kinase Inhibitors--pharmacology<br/>Proto-Oncogene Proteins c-bcl-2--metabolism<br/>Time Factors<br/>Transcription Factor RelA--metabolism<br/>Trimethyltin Compounds--toxicity<br/>X-Linked Inhibitor of Apoptosis Protein--metabolism<br/>p38 Mitogen-Activated Protein Kinases--metabolism