Doddrell, Robin D S <br/><br/>
Loss of SOX10 function contributes to the phenotype of human Merlin-null schwannoma cells.  [electronic resource] 

 -  Brain : a journal of neurology  Feb 2013 
 - 549-63 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1460-2156 
<br/><br/><label>Standard No.: </label>10.1093/brain/aws353  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Cells, Cultured<br/>Gene Knockdown Techniques<br/>Humans<br/>Mice<br/>Mice, Transgenic<br/>Neurilemmoma--genetics<br/>Neurofibromatosis 2--genetics<br/>Neurofibromin 2--deficiency<br/>Phenotype<br/>SOXE Transcription Factors--deficiency