Pottecher, Julien <br/><br/>
Cyclosporine A normalizes mitochondrial coupling, reactive oxygen species production, and inflammation and partially restores skeletal muscle maximal oxidative capacity in experimental aortic cross-clamping.  [electronic resource] 

 -  Journal of vascular surgery  Apr 2013 
 - 1100-1108.e2 p.  digital 
<br/><br/> Publication Type: Journal Article 
<br/><br/><label>ISSN: </label>1097-6809 
<br/><br/><label>Standard No.: </label>10.1016/j.jvs.2012.09.020  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Aorta--physiopathology<br/>Blotting, Western<br/>Calcium--metabolism<br/>Constriction<br/>Peptidyl-Prolyl Isomerase F<br/>Cyclophilins--metabolism<br/>Cyclosporine--administration & dosage<br/>Disease Models, Animal<br/>Electron Transport Chain Complex Proteins--metabolism<br/>Energy Metabolism--drug effects<br/>Immunohistochemistry<br/>Inflammation--metabolism<br/>Injections, Intraperitoneal<br/>Macrophages--drug effects<br/>Male<br/>Mitochondria, Heart--drug effects<br/>Mitochondria, Muscle--drug effects<br/>Mitochondrial Membrane Transport Proteins--antagonists & inhibitors<br/>Mitochondrial Permeability Transition Pore<br/>Muscle, Skeletal--blood supply<br/>Oxidative Stress--drug effects<br/>Rats<br/>Rats, Wistar<br/>Reactive Oxygen Species--metabolism<br/>Reperfusion Injury--metabolism<br/>Time Factors