Younce, Craig W
Exendin-4 attenuates high glucose-induced cardiomyocyte apoptosis via inhibition of endoplasmic reticulum stress and activation of SERCA2a. [electronic resource]
- American journal of physiology. Cell physiology Mar 2013
- C508-18 p. digital
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
ISSN: 1522-1563
Standard No.: 10.1152/ajpcell.00248.2012 doi
Subjects--Topical Terms: Animals Apoptosis--drug effects Calcium-Binding Proteins--metabolism Cells, Cultured Colforsin--pharmacology Diabetic Cardiomyopathies--metabolism Endoplasmic Reticulum Stress--drug effects Enzyme Activation Exenatide Glucagon-Like Peptide-1 Receptor Glucose--metabolism HSP70 Heat-Shock Proteins Heat-Shock Proteins--metabolism Hydrogen Peroxide--pharmacology Hyperglycemia--metabolism Hypoglycemic Agents--pharmacology Isoquinolines--pharmacology Membrane Proteins Myocytes, Cardiac--physiology Oxidative Stress--drug effects Peptide Fragments--pharmacology Peptides--pharmacology Phosphorylation--drug effects Protein Kinase Inhibitors--pharmacology RNA, Messenger--biosynthesis Rats Receptors, Glucagon--agonists Sarcoplasmic Reticulum Calcium-Transporting ATPases--antagonists & inhibitors Sulfonamides--pharmacology Thapsigargin--pharmacology Transcription Factor CHOP--biosynthesis Tunicamycin--pharmacology Unfolded Protein Response Venoms--pharmacology