Younce, Craig W

Exendin-4 attenuates high glucose-induced cardiomyocyte apoptosis via inhibition of endoplasmic reticulum stress and activation of SERCA2a. [electronic resource] - American journal of physiology. Cell physiology Mar 2013 - C508-18 p. digital

Publication Type: Journal Article; Research Support, Non-U.S. Gov't

1522-1563

10.1152/ajpcell.00248.2012 doi


Animals
Apoptosis--drug effects
Calcium-Binding Proteins--metabolism
Cells, Cultured
Colforsin--pharmacology
Diabetic Cardiomyopathies--metabolism
Endoplasmic Reticulum Stress--drug effects
Enzyme Activation
Exenatide
Glucagon-Like Peptide-1 Receptor
Glucose--metabolism
HSP70 Heat-Shock Proteins
Heat-Shock Proteins--metabolism
Hydrogen Peroxide--pharmacology
Hyperglycemia--metabolism
Hypoglycemic Agents--pharmacology
Isoquinolines--pharmacology
Membrane Proteins
Myocytes, Cardiac--physiology
Oxidative Stress--drug effects
Peptide Fragments--pharmacology
Peptides--pharmacology
Phosphorylation--drug effects
Protein Kinase Inhibitors--pharmacology
RNA, Messenger--biosynthesis
Rats
Receptors, Glucagon--agonists
Sarcoplasmic Reticulum Calcium-Transporting ATPases--antagonists & inhibitors
Sulfonamides--pharmacology
Thapsigargin--pharmacology
Transcription Factor CHOP--biosynthesis
Tunicamycin--pharmacology
Unfolded Protein Response
Venoms--pharmacology